Anti-Human MIP-1α (CCL3) (Polyclonal Rabbit) 0 ComentariosSubmit a Review Detalles del Producto Nùmero de Catalogo 500-P38 Descripcion Source: Polyclonal Rabbit Preparation: Produced from sera of rabbits pre-immunized with highly pure recombinant Human MIP-1α. Anti-Human MIP-1α specific antibody was purified by affinity chromatography employing immobilized Human MIP-1α matrix. Immunogen: E.coli derived Recombinant Human MIP-1α (CCL3) (PeproTech catalog# 300-08) Sandwich ELISA: To detect Human MIP-1α by sandwich ELISA (using 100 μl/well antibody solution) a concentration of 0.5 - 2.0 μg/ml of this antibody is required. This antigen affinity purified antibody, in conjunction with PeproTech’s Biotinylated Anti-Human MIP-1α (500-P38Bt) as a detection antibody, allows the detection of at least 0.2 - 0.4 ng/well of recombinant Human MIP-1α. Western Blot: To detect Human MIP-1α by Western Blot analysis this antibody can be used at a concentration of 0.1 - 0.2 μg/ml. When used in conjunction with compatible secondary reagents the detection limit for recombinant Human MIP-1α is 1.5 - 3.0 ng/lane, under either reducing or non-reducing conditions. Note: Additional applications tested on a lot-to-lot basis. Please contact Technical Support for more information. crossreactivity: Human References SDS Búsqueda del CoA Product Line Country Of Origin: USA Not for human use. Research Interest AIDS/HIV Angiogenesis/Cardiovascular Chemotaxis Immune System Inflammation Neurobiology Wound Healing Transplantation product.subtitle.recentcitations Primer autor Rius, C Titulo Trans- but not cis-resveratrol impairs angiotensin-II-mediated vascular inflammation through inhibition of NF-κB activation and peroxisome proliferator-activated receptor-gamma upregulation. Citar Journal of immunology (Baltimore, Md. : 1950); 185(6) pg3718-27 PudMed id 20709957 Primer autor Izhak, L Titulo Predominant expression of CCL2 at the tumor site of prostate cancer patients directs a selective loss of immunological tolerance to CCL2 that could be amplified in a beneficial manner. Citar Journal of immunology (Baltimore, Md. : 1950); 184(2) pg1092-101 PudMed id 19995900 Primer autor Pilette, C Titulo CCR4 ligands are up-regulated in the airways of atopic asthmatics after segmental allergen challenge. Citar The European Respiratory Journal; 23(6) pg876-84 PudMed id 15219001
